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Últimas novedades aparecidas en Renal Physiology

1	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-03-04* . [Traducción]
2	Eplerenone potentiates the antiproteinuric effects of enalapril in experimental nephrotic syndrome *Renal Physiology. Nephrotic syndrome (NS) is a clinical state characterized by massive proteinuria and edema. It is believed that nephrin and podocin are involved in the development of proteinuria. . 2008-03-04* . [Traducción]
3	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-03-03* . [Traducción]
4	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-03-03* . [Traducción]
5	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-03-03* . [Traducción]
6	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-03-03* . [Traducción]
7	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-03-03* . [Traducción]
8	Eplerenone potentiates the antiproteinuric effects of enalapril in experimental nephrotic syndrome *Renal Physiology. Nephrotic syndrome (NS) is a clinical state characterized by massive proteinuria and edema. It is believed that nephrin and podocin are involved in the development of proteinuria. . 2008-03-03* . [Traducción]
9	20-HETE-mediated cytotoxicity and apoptosis in ischemic kidney epithelial cells *Renal Physiology. These results demonstrate that 20-HETE overexpression can significantly exacerbate the cellular damage that is associated with renal IR injury and that the programmed cell death is mediated by activation of caspase-3 and is partially dependent on enhanced CYP4A generation of free radicals. . 2008-03-03* . [Traducción]
10	Amelioration of oxidative mitochondrial DNA damage and deletion after renal ischemic injury by the KATP channel opener diazoxide *Renal Physiology. We conclude that excess production of ROS by mitochondria on reperfusion is a major upstream event in renal reperfusion injury and that DZ functioned by preventing ROS accumulation in the mitochondria after I/R injury, thereby reducing oxidative stress as measured by the presence of oxidized mitochondrial DNA and features of apoptosis. . 2008-03-03* . [Traducción]
11	Bradykinin modulates focal adhesions and induces stress fiber remodeling in renal papillary collecting duct cells *Renal Physiology. Focal adhesions (FAs) are specialized regions of cell attachment to the extracellular matrix. Previous works have suggested that bradykinin (BK) can modulate cell-matrix interaction. In the present study, we used a physiological cellular model to evaluate the potential role of BK in modulating FAs and stress fibers. . 2008-03-03* . [Traducción]
12	Effects of targeted Bcl-2 expression in mitochondria or endoplasmic reticulum on renal tubular cell apoptosis *Renal Physiology. Collectively, the results suggest that the cytoprotective effects of Bcl-2 in different renal injury models are largely determined by its subcellular localizations. . 2008-03-03* . [Traducción]
13	Ezrin promotes functional expression and parathyroid hormone-mediated regulation of the sodium-phosphate cotransporter 2a in LLC-PK1 cells *Renal Physiology. The sodium-phosphate cotransporter 2a (NPT2a) is the principal phosphate transporter expressed in the brush border of renal proximal tubules and is downregulated by parathyroid hormone (PTH) through an endocytic mechanism. . 2008-03-03* . [Traducción]
14	Course of preeclamptic glomerular injury after delivery *Renal Physiology. We conclude that the functional manifestations of the glomerular endothelial injury of preeclampsia largely resolve within the first postpartum month. . 2008-02-29* . [Traducción]
15	Eplerenone potentiates the antiproteinuric effects of enalapril in experimental nephrotic syndrome *Renal Physiology. Nephrotic syndrome (NS) is a clinical state characterized by massive proteinuria and edema. It is believed that nephrin and podocin are involved in the development of proteinuria. . 2008-02-29* . [Traducción]
16	20-HETE-mediated cytotoxicity and apoptosis in ischemic kidney epithelial cells *Renal Physiology. These results demonstrate that 20-HETE overexpression can significantly exacerbate the cellular damage that is associated with renal IR injury and that the programmed cell death is mediated by activation of caspase-3 and is partially dependent on enhanced CYP4A generation of free radicals. . 2008-02-29* . [Traducción]
17	Amelioration of oxidative mitochondrial DNA damage and deletion after renal ischemic injury by the KATP channel opener diazoxide *Renal Physiology. We conclude that excess production of ROS by mitochondria on reperfusion is a major upstream event in renal reperfusion injury and that DZ functioned by preventing ROS accumulation in the mitochondria after I/R injury, thereby reducing oxidative stress as measured by the presence of oxidized mitochondrial DNA and features of apoptosis. . 2008-02-29* . [Traducción]
18	Bradykinin modulates focal adhesions and induces stress fiber remodeling in renal papillary collecting duct cells *Renal Physiology. Focal adhesions (FAs) are specialized regions of cell attachment to the extracellular matrix. Previous works have suggested that bradykinin (BK) can modulate cell-matrix interaction. In the present study, we used a physiological cellular model to evaluate the potential role of BK in modulating FAs and stress fibers. . 2008-02-29* . [Traducción]
19	Effects of targeted Bcl-2 expression in mitochondria or endoplasmic reticulum on renal tubular cell apoptosis *Renal Physiology. Collectively, the results suggest that the cytoprotective effects of Bcl-2 in different renal injury models are largely determined by its subcellular localizations. . 2008-02-29* . [Traducción]
20	Ezrin promotes functional expression and parathyroid hormone-mediated regulation of the sodium-phosphate cotransporter 2a in LLC-PK1 cells *Renal Physiology. The sodium-phosphate cotransporter 2a (NPT2a) is the principal phosphate transporter expressed in the brush border of renal proximal tubules and is downregulated by parathyroid hormone (PTH) through an endocytic mechanism. . 2008-02-29* . [Traducción]
21	Heparin-binding epidermal growth factor and Src family kinases in proliferation of renal epithelial cells *Renal Physiology. We suggest that RPTC proliferation after plating is mediated by HB-EGF produced through an autocrine/paracrine mechanism and RPTC proliferation following oxidant injury is mediated by Src without involvement of HB-EGF. . 2008-02-29* . [Traducción]
22	Inhibition of proinflammatory genes in anti-GBM glomerulonephritis by targeted dexamethasone-loaded AbEsel liposomes *Renal Physiology. This study demonstrates the potential of vascular bed-specific drug delivery aimed at disease-induced epitopes on the microvascular endothelial cells as a therapeutic strategy for glomerulonephritis. . 2008-02-29* . [Traducción]
23	Membrane current oscillations in descending vasa recta pericytes *Renal Physiology. We conclude that [Ca2+]CYT oscillations stimulate Cl– currents. The former are most likely maintained by repetitive cycles of ryanodine-sensitive SR Ca2+ release and SKF96365-sensitive store refilling. . 2008-02-29* . [Traducción]
24	Protective effects of low-dose carbon monoxide against renal fibrosis induced by unilateral ureteral obstruction *Renal Physiology. Our findings suggest that low-dose CO exerts protective effects, via the MKK3 pathway, to inhibit development of renal fibrosis in obstructive nephropathy. . 2008-02-29* . [Traducción]
25	Restoration of CREB function ameliorates cisplatin cytotoxicity in renal tubular cells *Renal Physiology. We have shown that mouse proximal tubule cells (TKPTS) survive H2O2 stress by activating the cAMP-responsive element binding protein (CREB)-mediated transcription via the canonical EGFR-Ras/ERK pathway. . 2008-02-29* . [Traducción]

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